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Oriented immobilized anti-hIgG via F(c) fragment-imprinted PHEMA cryogel for IgG purification
Isolation and some properties of an IgG Fc-binding protein from group A streptococci type 15
Surgical outcomes in patients with haemophilia A or B receiving extended half-life recombinant factor VIII and IX Fc fusion proteins : Real-world experience in the Nordic countries
Interaction of the Fc part of IgG with Lancefield extracts of hemolytic streptococci. Strain specificity and activity
Herpes simplex type 1-induced Fc receptor binds to the Cgamma2-Cgamma3 interface region of IgG in the area that binds staphylococcal protein A
Identification of the site on IgG Fc for interaction with streptococci of groups A, C and G
Biological and structural characterization of murine TRALI antibody reveals increased Fc-mediated complement activation
Transfusion-related acute lung injury (TRALI) remains a leading cause of transfusionrelated deaths. In most cases, anti-leukocyte antibodies in the transfusion product trigger TRALI, but not all anti-leukocyte antibodies cause TRALI. It has been shown that the anti-major histocompatibility complex (MHC) class I antibody 34-1-2S (anti-H-2Kd) causes TRALI in BALB/c mice (MHC class I haplotype H-2Kd)
Interaction between herpes simplex type 1-induced Fc receptor and human and rabbit immunoglobulin G (IgG) domains
Studies of protein A and herpes simplex virus-1 induced Fc gamma-binding specificities. Different binding patterns for IgG3 from Caucasian and Oriental subjects
The Functional Polymorphism 844 A>G in Fc{alpha}RI (CD89) Does Not Contribute to Systemic Sclerosis or Rheumatoid Arthritis Susceptibility.
Absence of host-cell influence on binding specificity of herpes simplex virus type 1 induced Fc receptor
Specificity of Fc receptors induced by herpes simplex virus type 1: comparison of immunoglobulin G from different animal species
Subclass-switched anti-Spike IgG3 oligoclonal cocktails strongly enhance Fc-mediated opsonization
Human immunoglobulin class and subclass specificity of Fc receptors induced by herpes simplex virus type 1
Binding of human and animal immunoglobulins to the IgG Fc receptor induced by human cytomegalovirus
Characterization of herpes simplex virus type 1-induced Fc receptor in its interaction with rabbit immunoglobulin G (IgG)
Platelet Activation and Aggregation Induced by Streptococcus bovis/Streptococcus equinus Complex
Streptococcus bovis/Streptococcus equinus complex (SBSEC) is a common cause of infective endocarditis (IE). For IE-pathogens, the capacity to activate and aggregate platelets is believed to be an important virulence mechanism. While the interactions between bacteria and platelets have been described in detail for many Gram-positive pathogens, little research has been carried out with SBSEC in this
