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Microsoft Word - LIVD02 litteraturlista VT26
Microsoft Word - LIVR37_litteraturlista_VT26_
COP26 Event: How Climate Change and Human Rights are Connected - The Raoul Wallenberg Institute of H
Litteraturlista FTEB06 - VT26
COP26, climate justice and human rights: towards a grounds-up international law? - The Raoul Wallenb
Numerical computations of steady and unsteady flow in bended pipes
Litteraturlista KOGB01 - VT26
Litteraturlista KOGP06 - VT26
MRSN03_Literature_Spring26
Online Symposium on Human Rights in Transition 25-26 October - The Raoul Wallenberg Institute of Hum
Litteraturlista KOGP06 - VT26
Microsoft Word - LIVR03 litteraturlista VT26
Litteraturlista KOGB01 - VT26
MRSN03_Literature_Spring26
Microsoft Word - 2008-08-26 - Marklund-Lundin - Learning using computer based simulation.doc
Choosing your optional courses
Choosing Your Optional Courses! General Information: Within the BIDS programme, each student has the opportunity to study optional courses equaling 30 ECTS in order to either further specialise within your chosen major or broaden your degree, see example below: Student 1: Chooses to major in political science. In year 2, the student starts by studying 30 ECTS at the department of Political Science
https://www.keg.lu.se/sites/keg.lu.se/files/choosing_your_optional_courses.pdf - 2026-06-07
Campuskarta-2024-eng
N-glycans and the N terminus of protein C inhibitor affect the cofactor-enhanced rates of thrombin inhibition
Protein C inhibitor (PCI) is a serine protease inhibitor, displaying broad protease specificity, found in blood and other tissues. In blood, it is capable of inhibiting both procoagulant and anticoagulant proteases. Mechanisms that provide specificity to PCI remain largely unrevealed. In this study we have for the first time provided a full explanation for the marked size heterogeneity of blood-de
Four modes of adhesion are used during Helicobacter pylori binding to human mucins in the oral and gastric niches
Background: Helicobacter pylori causes peptic ulcer disease and gastric cancer, and the oral cavity is likely to serve as a reservoir for this pathogen. We investigated the binding of H. pylori to the mucins covering the mucosal surfaces in the niches along the oral to gastric infection route and during gastric disease and modeled the outcome of these interactions. Materials and Methods: A panel o
