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Differentiation of myeloid leukemic cells in vitro demonstrated by microcalorimetry : Stimulation of leukemic and remission cells by IgG-binding Fc receptors
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No association of primary Sjogren's syndrome with Fc gamma receptor gene variants
Transiently binding antibody fragments against Lewis x and sialyl-Lewis x
Biomolecular recognition is often characterised by low affinity where many weak interactions work either alone or in concert, resulting in an inherent dynamic situation. For example the well-studied weak binding of cell-cell interactions is predominantly based on a range of carbohydrates that interact with numerous (protein) ligands. Finding appropriate binders to these carbohydrate structures may
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Bacterial Modulation of Fc Effector Functions
Oriented immobilized anti-hIgG via F(c) fragment-imprinted PHEMA cryogel for IgG purification
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Isolation and some properties of an IgG Fc-binding protein from group A streptococci type 15
Interaction of the Fc part of IgG with Lancefield extracts of hemolytic streptococci. Strain specificity and activity
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Human immunoglobulin class and subclass specificity of Fc receptors induced by herpes simplex virus type 1
Absence of host-cell influence on binding specificity of herpes simplex virus type 1 induced Fc receptor
Biological and structural characterization of murine TRALI antibody reveals increased Fc-mediated complement activation
Transfusion-related acute lung injury (TRALI) remains a leading cause of transfusionrelated deaths. In most cases, anti-leukocyte antibodies in the transfusion product trigger TRALI, but not all anti-leukocyte antibodies cause TRALI. It has been shown that the anti-major histocompatibility complex (MHC) class I antibody 34-1-2S (anti-H-2Kd) causes TRALI in BALB/c mice (MHC class I haplotype H-2Kd)