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Molekylär neuromodulering

Fascineras du också av hjärnan och dess komplexitet men också av hur nya tekniker och den snabba utvecklingen inom bioteknologi kan öppna upp för helt nya behandlingar? Då kan vår forskning vara något för dig! Vårt fokus är på att utveckla avancerade terapier såsom cell- och genterapi för Parkinsons sjukdom. Vi arbetar också hårt för att hitta ett botemedel för en av de mest fruktansvärda sjukdomaAre you intrigued by the brain and its complexity, as well as by new technologies and the amazing developments in biotechnology? Then our research may be for you. Our main focus is on developing advanced therapeutical medical products (ATMPs), such as cell and gene therapy for Parkinson's disease. We are also working to treat one of the most devastating disorders of the human brain: glioblastomas,

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We sought to determine whether specific characteristics of vertebral fractures in elderly men are associated with low bone mineral density (BMD) and osteoporosis. Mister osteoporosis Sweden is a population based cohort study involving 3014 men aged 69 to 81 years. Of these, 1427 had readable lateral radiographs of the thoracic and lumbar spine. Total body (TB) BMD (g/cm²) and total right hip (TH)

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Concordant with a wider political and cultural emphasis on individual choice and selfcare, European patients have during the last decade become empowered stakeholders as a result of digitization. Nonetheless, patient empowerment sits uncomfortable with the prevailing business model pertaining to the majority of pharmaceutical organisations whose PR activities primarily target healthcare profession

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Background: With the rapid development of new genetic measurement methods, several types of genetic alterations can be quantified in a high-throughput manner. While the initial focus has been on investigating each data set separately, there is an increasing interest in studying the correlation structure between two or more data sets. Multivariate methods based on Canonical Correlation Analysis (CC

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Objectives In rheumatoid arthritis (RA), seropositive and seronegative disease may be two entities with different underlying pathophysiological mechanisms, long-term outcomes and disease presentations. However, the effect of the conjoint presence of multiple autoantibodies, as proxy for a more pronounced humoral autoimmune response, on clinical phenotype remains unclear. Therefore, this study inve

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